The information on this website is not intended for diagnosis or treatment. Please consult your physician for health concerns.

Skip to main content

Treatment-Resistant Depression — Ketamine, Esketamine, and Rapid-Acting Antidepressants

Comprehensive management of treatment-resistant depression including modern definition criteria, intravenous racemic ketamine and intranasal esketamine therapy, monitoring protocols, and emerging psychedelic-assisted approaches for severe and refractory mood disorders.

Written by: Saygı Hospital Health Guide Editorial Board
Last updated:

This content has been compiled by the Saygı Hospital Health Guide Editorial Board and is periodically reviewed by a specialist physician.

References (5)

This content is for informational purposes only and does not constitute medical advice. You can book an appointment at our Psikiyatri department. Book Appointment →

What is Treatment-Resistant Depression — Ketamine, Esketamine, and Rapid-Acting Antidepressants?

Treatment-resistant depression (TRD) refers to major depressive disorder (MDD) that has not responded adequately to standard antidepressant treatments. While definitions vary, the most widely accepted criterion is failure to achieve adequate response (≥50% reduction in depressive symptoms) after at least two adequate trials of antidepressants from different mechanistic classes, given for sufficient duration (typically 6-8 weeks each) at therapeutic doses. TRD affects 30-40% of patients with MDD and is associated with substantial morbidity, mortality (particularly suicide), functional impairment, and economic burden. Risk factors include early age of onset, comorbid anxiety, substance use disorders, medical comorbidity, suicidal ideation, and certain personality disorders.

Ketamine, originally developed as a general anesthetic, was discovered to have rapid and profound antidepressant effects through NMDA receptor antagonism, AMPA receptor activation, increased glutamate release in the prefrontal cortex, BDNF upregulation, and synaptic plasticity enhancement. Unlike traditional monoamine-based antidepressants requiring weeks for full effect, ketamine produces antidepressant effects within hours, with peak response at 24 hours and sustained effects for 1-2 weeks. Intravenous racemic ketamine (0.5 mg/kg over 40 minutes) has been used off-label, while the S-enantiomer esketamine intranasal spray (Spravato) received FDA approval in 2019 specifically for TRD in conjunction with an oral antidepressant.

Modern protocols include induction phase with twice-weekly dosing for 4 weeks, followed by maintenance phase with weekly or biweekly administration based on response, with strict monitoring requirements including REMS (Risk Evaluation and Mitigation Strategy) program enrollment for esketamine. During and after each session, patients require monitoring for dissociative effects (typically peak 40 minutes post-dose, resolving within 2 hours), elevated blood pressure, sedation, and rare but serious effects including suicidal ideation, hepatotoxicity, and cystitis. Patients should not drive or operate machinery for the rest of treatment day. Comprehensive evaluation prior to initiation includes psychiatric assessment, suicide risk evaluation, substance use history, cardiovascular evaluation (especially with elevated BP), and discussion of benefits and risks. Emerging therapies include psilocybin-assisted therapy (FDA breakthrough therapy designation), MDMA-assisted therapy for PTSD, dextromethorphan-bupropion (Auvelity, FDA-approved 2022), and continued investigation of other novel mechanisms.

Symptoms

Persistent depressed mood despite adequate antidepressant trials
Inadequate response to ≥2 antidepressant classes (TRD definition)
Severe functional impairment in work and relationships
Suicidal ideation or behavior despite treatment
Anhedonia, fatigue, sleep disturbance, appetite changes
Cognitive symptoms: poor concentration, indecisiveness, hopelessness
Comorbid anxiety, substance use, medical conditions

Risk Factors

Early age of MDD onset
Comorbid anxiety disorders or substance use
Childhood trauma or adverse experiences
Family history of treatment-resistant depression
Comorbid medical conditions (especially chronic pain)
Personality disorders (especially borderline)
Suicidal history or current suicidal ideation

When to See a Doctor?

If you experience any of the following symptoms, seek medical attention promptly:

  • Inadequate response to two antidepressant trials
  • Severe depression with suicidal ideation
  • Functional impairment despite ongoing treatment
  • Need for rapid antidepressant effect
  • Treatment-resistant symptoms requiring specialist evaluation
  • Consideration of ketamine or esketamine therapy
  • Comprehensive psychiatric evaluation for TRD diagnosis

Treatment Methods

01
Intranasal esketamine (Spravato) with oral antidepressant per REMS protocol
02
Intravenous ketamine 0.5 mg/kg over 40 minutes (off-label, twice weekly induction)
03
ECT (electroconvulsive therapy) for severe TRD or with psychotic features
04
Augmentation strategies: lithium, atypical antipsychotics, T3 thyroid
05
Combination antidepressants (different mechanism classes)
06
Transcranial magnetic stimulation (TMS) for unilateral or bilateral protocols
07
Emerging: psilocybin-assisted therapy, dextromethorphan-bupropion, vagal nerve stimulation

Which Department to Visit?

You can visit our Psikiyatri department for these complaints. Our specialist physicians will create the most suitable treatment plan for you.

Learn About Psikiyatri Department

Let us help you

You can make an appointment with our specialists or contact us for your concerns.

Health Disclaimer: The information on this page is prepared for general informational purposes only. It does not replace medical diagnosis and treatment. Please consult your physician for your complaints. Saygı Hospital does not accept responsibility for actions taken based on the information on this page.