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Rhino-Orbital-Cerebral Mucormycosis

Rapidly progressive, high-mortality necrotizing fungal infection

Written by: Saygı Hospital Health Guide Editorial Board
Last updated:

This content has been compiled by the Saygı Hospital Health Guide Editorial Board and is periodically reviewed by a specialist physician.

References (5)

This content is for informational purposes only and does not constitute medical advice. You can book an appointment at our Internal Medicine department. Book Appointment →

What is Rhino-Orbital-Cerebral Mucormycosis?

Mucormycosis is an invasive, angioinvasive fungal infection caused by ubiquitous environmental molds of the order Mucorales (Rhizopus, Mucor, Lichtheimia, Cunninghamella, Apophysomyces). The rhino-orbital-cerebral form begins with spore inhalation and germination in nasal turbinates and paranasal sinuses, followed by invasion of arterial walls causing thrombosis, tissue infarction, and contiguous spread to orbit and brain through ethmoid plates, cribriform plate, or cavernous sinus. Risk factors include diabetic ketoacidosis (most common globally), hematologic malignancies and stem cell transplant with profound neutropenia, deferoxamine therapy increasing free iron, severe COVID-19 with corticosteroid use, and rarely solid organ transplantation, AIDS, and major trauma.

Initial symptoms include facial pain and swelling, nasal congestion with bloody discharge, periorbital pain, and headache, often progressing within hours to days. Examination may reveal black nasal eschar, palatal necrosis, eyelid swelling, proptosis, ophthalmoplegia, and decreased visual acuity. Cerebral involvement causes altered mental status, seizures, and focal deficits. Imaging with CT and MRI shows pansinusitis with bone erosion, orbital extension with extraocular muscle and optic nerve involvement, cavernous sinus thrombosis, and brain infarcts or abscesses. Diagnosis requires endoscopic biopsy with histopathology demonstrating broad, ribbon-like, non-septate hyphae with right-angle branching invading vessels and tissue, supplemented by fungal culture and PCR.

Mortality without aggressive treatment exceeds 50-80%. Therapy combines emergency reversal of underlying immunosuppression (insulin therapy and ketoacidosis correction in DKA, granulocyte colony stimulating factor for neutropenia, corticosteroid taper), wide surgical debridement of all necrotic tissue including orbital exenteration when needed, and prolonged antifungal therapy with liposomal amphotericin B (5-10 mg/kg/day) for 4-6 weeks followed by oral isavuconazole or posaconazole step-down for 3-6 months. Adjunctive measures include hyperbaric oxygen, iron chelation with deferasirox, and aggressive supportive care. Survivors often have major facial deformities and visual loss requiring complex reconstruction.

Symptoms

Rapidly worsening facial or periorbital pain
Black necrotic eschar in nasal cavity or palate
Bloody nasal discharge with sinus tenderness
Eyelid swelling and proptosis
Ophthalmoplegia and vision loss
Headache and altered mental status
Fever and rapid clinical deterioration

Risk Factors

Diabetic ketoacidosis or poorly controlled diabetes
Hematologic malignancy with neutropenia
Hematopoietic stem cell or solid organ transplantation
Severe COVID-19 with corticosteroid use
Iron overload and deferoxamine therapy
Prolonged corticosteroid or cytotoxic chemotherapy
Major trauma or burns with environmental contamination

When to See a Doctor?

If you experience any of the following symptoms, seek medical attention promptly:

  • Severe sinus pain with facial swelling in immunocompromised patient
  • Black eschar in nasal cavity or on palate
  • New proptosis or vision change with sinus symptoms
  • Periorbital cellulitis unresponsive to standard antibiotics
  • Altered mental status with sinusitis in diabetic ketoacidosis
  • Suspected fungal infection during severe COVID-19 hospitalization
  • Rapid clinical deterioration in immunosuppressed patient

Treatment Methods

01
Urgent CT and MRI of sinuses, orbit, and brain
02
Endoscopic biopsy with histopathology and fungal culture
03
Liposomal amphotericin B 5-10 mg/kg/day intravenously
04
Step-down therapy with isavuconazole or posaconazole
05
Wide surgical debridement, orbital exenteration if indicated
06
Reversal of underlying immunosuppression
07
Adjunctive hyperbaric oxygen and iron chelation

Which Department to Visit?

You can visit our Enfeksiyon Hastalıkları department for these complaints. Our specialist physicians will create the most suitable treatment plan for you.

Learn About Enfeksiyon Hastalıkları Department

Let us help you

You can make an appointment with our specialists or contact us for your concerns.

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Health Disclaimer: The information on this page is prepared for general informational purposes only. It does not replace medical diagnosis and treatment. Please consult your physician for your complaints. Saygı Hospital does not accept responsibility for actions taken based on the information on this page.