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Diabetic Foot Prevention Strategies

Evidence-based multidisciplinary approach to preventing diabetic foot ulceration, infection, and amputation through risk stratification, education, footwear optimization, and structured care pathways.

Written by: Saygı Hospital Health Guide Editorial Board
Last updated:

This content has been compiled by the Saygı Hospital Health Guide Editorial Board and is periodically reviewed by a specialist physician.

References (5)

This content is for informational purposes only and does not constitute medical advice. You can book an appointment at our Endokrinoloji department. Book Appointment →

What is Diabetic Foot Prevention Strategies?

Risk stratification and pathophysiology: 1) IWGDF (International Working Group on Diabetic Foot) risk categories - Category 0: no neuropathy or PAD (annual screening); Category 1: neuropathy without deformity (6-monthly); Category 2: neuropathy with deformity or PAD (3-4 monthly); Category 3: history of ulcer or amputation (1-3 monthly); 2) Pathogenesis - peripheral neuropathy (sensory, motor, autonomic) leads to loss of protective sensation, foot deformity (claw toes, Charcot foot), dry skin with fissures; peripheral artery disease (PAD) reduces healing capacity; trauma from footwear, foreign objects, burns initiates injury; impaired immune function and hyperglycemia delay healing; 3) Charcot neuroarthropathy - progressive non-infectious destructive arthropathy in neuropathic foot, often misdiagnosed as cellulitis; midfoot collapse leads to rocker-bottom deformity and ulceration; 4) Foot deformities - hallux valgus, hammer toes, prominent metatarsal heads, equinus contracture, post-amputation altered mechanics increase pressure points; 5) Vascular - PAD diagnosed by ABI <0.9, toe pressure <30 mmHg, TcPO2 <30 mmHg suggest critical limb ischemia.

Education, examination, and footwear: 1) Patient education - daily foot self-examination including visual inspection (mirror for plantar surface) and palpation; never walk barefoot; check shoes for foreign objects before wearing; wash feet daily and dry thoroughly especially between toes; moisturize but not between toes; cut nails straight across; do not perform bathroom surgery on calluses or corns; report any new lesion immediately; 2) Annual comprehensive foot examination - 10g monofilament test (4-10 sites), vibration testing (128 Hz tuning fork), Achilles reflex, dorsal pedis and posterior tibial pulses, ABI if PAD suspected, skin/nail/deformity inspection, footwear assessment; 3) Footwear principles - extra-depth shoes with rocker bottoms for high-risk patients, custom orthoses to redistribute pressure, never wear slippers or flip-flops outdoors, replace shoes every 6-12 months, professional fitting; 4) Therapeutic footwear in high-risk - off-loading footwear (post-op shoes, total contact casts for active ulcers), accommodative orthoses with custom inserts; reduce ulcer recurrence 50%; 5) Prophylactic surgery - tendon lengthening for equinus, exostectomy for prominent bones, claw toe correction in selected high-risk cases.

Multidisciplinary care and risk modification: 1) Multidisciplinary foot clinic - includes diabetologist, podiatrist, vascular surgeon, infectious disease, plastic surgery, orthotist, diabetic nurse educator; reduces amputation 50-85%; 2) Glycemic control - HbA1c <7% in most, individualized; tight control reduces neuropathy progression; 3) Cardiovascular risk factor management - statin, ACEi/ARB, antiplatelet (aspirin or clopidogrel), blood pressure <130/80; 4) Smoking cessation - critical, more than doubles amputation risk in PAD; 5) Vascular intervention - endovascular or surgical revascularization for critical limb ischemia, ABI <0.5, severe rest pain, non-healing ulcer; 6) Skin and nail care - regular podiatric debridement of calluses (reduces pressure 25%), nail cutting by professional in high-risk; 7) Specific high-risk groups - chronic kidney disease (especially dialysis) markedly increases risk and mortality; post-transplant patients; previous amputation; 8) Telemedicine and remote monitoring - thermal foot mats, smart insoles emerging for early ulcer detection; foot temperature differences >2.2°C predict ulceration; 9) Cost-effectiveness - prevention is cost-effective; structured programs save 2-5x cost of care.

Symptoms

Loss of protective sensation in feet
Tingling, burning, or numbness
Foot deformity (claw toes, hallux valgus)
Dry skin with fissures, callus formation
Cold feet, absent pulses (PAD)
Any new wound, blister, or ulcer

Risk Factors

Long-standing diabetes (>10 years)
Poor glycemic control (HbA1c >8%)
Peripheral neuropathy (loss of sensation)
Peripheral artery disease, smoking
Previous foot ulcer or amputation
Chronic kidney disease, dialysis

When to See a Doctor?

If you experience any of the following symptoms, seek medical attention promptly:

  • Annual foot examination for all diabetics
  • Any new wound, blister, or color change
  • Increased temperature, swelling, redness
  • Loss of sensation or new tingling
  • Foot deformity or shoe fitting problems
  • Infection signs (pus, fever, increased pain)

Treatment Methods

01
Risk stratification and tailored screening
02
Daily foot self-exam and patient education
03
Therapeutic footwear with custom orthoses
04
Multidisciplinary foot clinic referral
05
Prophylactic podiatric care (callus, nails)
06
Glycemic, vascular, and smoking optimization

Which Department to Visit?

You can visit our Endokrinoloji department for these complaints. Our specialist physicians will create the most suitable treatment plan for you.

Learn About Endokrinoloji Department

Let us help you

You can make an appointment with our specialists or contact us for your concerns.

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Health Disclaimer: The information on this page is prepared for general informational purposes only. It does not replace medical diagnosis and treatment. Please consult your physician for your complaints. Saygı Hospital does not accept responsibility for actions taken based on the information on this page.